Exercise performance and V0₂ kinetics during upright and recumbent high-intensity cycling exercise

Egana, Mikel, O'Riordan, Damien and Warmington, Stuart A. 2010, Exercise performance and V0₂ kinetics during upright and recumbent high-intensity cycling exercise, European journal of applied physiology, vol. 110, no. 1, pp. 39-47.

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Title Exercise performance and V0₂ kinetics during upright and recumbent high-intensity cycling exercise
Author(s) Egana, Mikel
O'Riordan, Damien
Warmington, Stuart A.
Journal name European journal of applied physiology
Volume number 110
Issue number 1
Start page 39
End page 47
Total pages 9
Publisher Springer
Place of publication Heidelberg, Germany
Publication date 2010-09
ISSN 1439-6319
1439-6327
Keyword(s) Recumbent
Cycling
Posture
Performance
VO₂ kinetics
Summary This study investigated cycling performance and oxygen uptake (VO2) kinetics between upright and two commonly used recumbent (R) postures, 65ºR and 30ºR. On three occasions, ten young active males performed three bouts of high-intensity constant-load (85% peak workload achieved during a graded test) cycling in one of the three randomly assigned postures (upright, 65ºR or 30ºR). The first bout was performed to fatigue and second and third bouts were limited to 7 min. A subset of seven subjects performed a final constant-load test to failure in the supine posture. Exercise time to failure was not altered when the body inclination was lowered from the upright (13.1 ± 4.5 min) to 65ºR (10.5 ± 2.7 min) and 30ºR (11.5 ± 4.6 min) postures; but it was significantly shorter in the supine posture (5.8 ± 2.1 min) when compared with the three inclined postures. Resulting kinetic parameters from a tri-exponential analysis of breath-by-breath VO2 data during the first 7 min of exercise were also not different between the three inclined postures. However, inert gas rebreathing analysis of cardiac output revealed a greater cardiac output and stroke volume in both recumbent postures compared with the upright posture at 30 s into the exercise. These data suggest that increased cardiac function may counteract the reduction of hydrostatic pressure from upright ~25 mmHg; to 65ºR ~22 mmHg; and 30ºR ~18 mmHg such that perfusion of active muscle presumably remains largely unchanged, and also therefore, VO2 kinetics and performance during high-intensity cycling.
Notes Published online: 13 April 2010
Language eng
Field of Research 110602 Exercise Physiology
Socio Economic Objective 970106 Expanding Knowledge in the Biological Sciences
HERDC Research category C1 Refereed article in a scholarly journal
HERDC collection year 2010
Copyright notice ©2010, Springer-Verlag
Persistent URL http://hdl.handle.net/10536/DRO/DU:30030460

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Created: Wed, 13 Oct 2010, 10:29:30 EST by Jane Moschetti

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