Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathways

White, Anthony R., Huang, Xudong, Jobling, Michael F., Barrow, Colin J., Beyreuther, Konrad, Masters, Colin L., Bush, Ashley I. and Cappai, Roberto 2001, Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathways, Journal of neurochemistry, vol. 76, no. 5, pp. 1509-1520, doi: 10.1046/j.1471-4159.2001.00178.x.

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Title Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathways
Author(s) White, Anthony R.
Huang, Xudong
Jobling, Michael F.
Barrow, Colin J.ORCID iD for Barrow, Colin J. orcid.org/0000-0002-2153-7267
Beyreuther, Konrad
Masters, Colin L.
Bush, Ashley I.
Cappai, Roberto
Journal name Journal of neurochemistry
Volume number 76
Issue number 5
Start page 1509
End page 1520
Total pages 12
Publisher Wiley-Blackwell
Place of publication Chichester, Eng.
Publication date 2001-03
ISSN 0022-3042
Keyword(s) Alzheimer Disease
Amyloid beta-Protein Precursor
Animals
Cell Survival
Cells, Cultured
Cerebral Cortex
Copper
Dose-Response Relationship, Drug
Drug Synergism
Homocysteine
Humans
Kinetics
Mice
Mice, Inbred Strains
Neurons
Neurotoxins
Risk Factors
Language eng
DOI 10.1046/j.1471-4159.2001.00178.x
Field of Research 1109 Neurosciences
HERDC Research category C1.1 Refereed article in a scholarly journal
Copyright notice ©2001, International Society for Neurochemistry
Persistent URL http://hdl.handle.net/10536/DRO/DU:30099782

Document type: Journal Article
Collection: School of Life and Environmental Sciences
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