MYCN amplification drives an aggressive form of spinal ependymoma

Ghasemi, David R., Sill, Martin, Okonechnikov, Konstantin, Korshunov, Andrey, Yip, Stephen, Schutz, Peter W., Scheie, David, Kruse, Anders, Harter, Patrick N., Kastelan, Marina, Wagner, Marlies, Hartmann, Christian, Benzel, Julia, Maass, Kendra K., Khasraw, Mustafa, Sträter, Ronald, Thomas, Christian, Paulus, Werner, Kratz, Christian P., Witt, Hendrik, Kawauchi, Daisuke, Herold-Mende, Christel, Sahm, Felix, Brandner, Sebastian, Kool, Marcel, Jones, David T.W., von Deimling, Andreas, Pfister, Stefan M., Reuss, David E. and Pajtler, Kristian W. 2019, MYCN amplification drives an aggressive form of spinal ependymoma, Acta Neuropathologica, pp. 1-15, doi: 10.1007/s00401-019-02056-2.

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Title MYCN amplification drives an aggressive form of spinal ependymoma
Author(s) Ghasemi, David R.
Sill, Martin
Okonechnikov, Konstantin
Korshunov, Andrey
Yip, Stephen
Schutz, Peter W.
Scheie, David
Kruse, Anders
Harter, Patrick N.
Kastelan, Marina
Wagner, Marlies
Hartmann, Christian
Benzel, Julia
Maass, Kendra K.
Khasraw, MustafaORCID iD for Khasraw, Mustafa orcid.org/0000-0003-3249-9849
Sträter, Ronald
Thomas, Christian
Paulus, Werner
Kratz, Christian P.
Witt, Hendrik
Kawauchi, Daisuke
Herold-Mende, Christel
Sahm, Felix
Brandner, Sebastian
Kool, Marcel
Jones, David T.W.
von Deimling, Andreas
Pfister, Stefan M.
Reuss, David E.
Pajtler, Kristian W.
Journal name Acta Neuropathologica
Start page 1
End page 15
Total pages 15
Publisher Springer
Place of publication Berlin, Germany
Publication date 2019
ISSN 0001-6322
1432-0533
Keyword(s) Ependymoma
Intradural extramedullary ependymoma
MYCN
Spinal tumor
DNA methylation
CNS
malignancies
Notes In Press
Language eng
DOI 10.1007/s00401-019-02056-2
Indigenous content off
Field of Research 1103 Clinical Sciences
1109 Neurosciences
HERDC Research category C1 Refereed article in a scholarly journal
Persistent URL http://hdl.handle.net/10536/DRO/DU:30129564

Document type: Journal Article
Collections: Faculty of Health
School of Medicine
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