Attenuating PI3K/Akt- mTOR pathway reduces dihydrosphingosine 1 phosphate mediated collagen synthesis and hypertrophy in primary cardiac cells
Version 2 2024-06-05, 10:28Version 2 2024-06-05, 10:28
Version 1 2021-11-23, 08:12Version 1 2021-11-23, 08:12
journal contribution
posted on 2024-06-05, 10:28 authored by RR Magaye, Feby SaviraFeby Savira, Y Hua, X Xiong, L Huang, C Reid, BL Flynn, D Kaye, D Liew, BH WangAttenuating PI3K/Akt- mTOR pathway reduces dihydrosphingosine 1 phosphate mediated collagen synthesis and hypertrophy in primary cardiac cells
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Journal
International Journal of Biochemistry and Cell BiologyVolume
134Article number
ARTN 105952Pagination
1 - 16Location
NetherlandsPublisher DOI
ISSN
1357-2725eISSN
1878-5875Language
EnglishPublication classification
C1 Refereed article in a scholarly journalPublisher
PERGAMON-ELSEVIER SCIENCE LTDUsage metrics
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Keywords
Science & TechnologyLife Sciences & BiomedicineBiochemistry & Molecular BiologyCell BiologyFibrosisHypertrophyDihydrosphingosine 1 phosphateSphingolipidPI3KProtein kinase BAktmTORRibosomal protein S6Cardiac remodellingGLYCOGEN-SYNTHASE KINASE-3-BETARECEPTOR STIMULATIONSPHINGOSINE-1-PHOSPHATEHEARTPHOSPHORYLATIONTRANSCRIPTIONGATA4MICEFIBROBLASTSINHIBITION3101 Biochemistry and cell biology
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