Loss of protein kinase D activity demonstrates redundancy in cardiac glucose metabolism and preserves cardiac function in obesity

Version 3 2024-06-18, 23:49

Version 2 2024-06-04, 03:33

Version 1 2020-10-26, 08:12

journal contribution

posted on 2024-06-18, 23:49 authored by KA De Jong, LG Hall, MC Renton, Timothy ConnorTimothy Connor, SD Martin, Greg KowalskiGreg Kowalski, Chris ShawChris Shaw, Clinton BruceClinton Bruce, Kirsten HowlettKirsten Howlett, Sean McgeeSean Mcgee

Loss of protein kinase D activity demonstrates redundancy in cardiac glucose metabolism and preserves cardiac function in obesity

History

Journal

Molecular Metabolism

Volume

42

Article number

101105

Pagination

1 - 11

Location

Germany

Publisher DOI

https://doi.org/10.1016/j.molmet.2020.101105

Open access

Yes

Link to full text

http://doi.org/10.1016/j.molmet.2020.101105

ISSN

2212-8778

eISSN

2212-8778

Language

English

Author URL

http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000598345600007&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=a045e4b2bb1f2b747c68c720ec8913b7

Publication classification

C1 Refereed article in a scholarly journal

Publisher

ELSEVIER

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Categories

Keywords

Science & Technology Life Sciences & Biomedicine Endocrinology & Metabolism Protein kinase D Cardiac glucose metabolism Stable isotope metabolomics Cardiac function Obesity Metabolic redundancy 3101 Biochemistry and cell biology 3201 Cardiovascular medicine and haematology 3202 Clinical sciences

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NLM

DC