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Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS
journal contribution
posted on 2015-11-01, 00:00 authored by Kai Y Soo, Mark Halloran, Vinod SundaramoorthyVinod Sundaramoorthy, Sonam Parakh, Reka P Toth, Katherine A Southam, Catriona A McLean, Peter Lock, Anna King, Manal A Farg, Julie D AtkinRab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS
History
Journal
Acta NeuropathologicaVolume
130Issue
5Pagination
679 - 697Publisher
SpringerLocation
Berlin, GermanyPublisher DOI
ISSN
0001-6322eISSN
1432-0533Language
engPublication classification
C1 Refereed article in a scholarly journalUsage metrics
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No categories selectedKeywords
Science & TechnologyLife Sciences & BiomedicineClinical NeurologyNeurosciencesPathologyNeurosciences & NeurologyTDP-43FUSSOD1ER-Golgi transportAmyotrophic lateral sclerosisAMYOTROPHIC-LATERAL-SCLEROSISGTP-BINDING PROTEINENDOPLASMIC-RETICULUMSUPEROXIDE-DISMUTASEMOTOR-NEURONSINTERMEDIATE COMPARTMENTSELECTIVE VULNERABILITYCOAT PROTEINSCELL-SURFACESPINAL-CORDER–Golgi transport
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