Schizophrenia is primed for an increased expression of depression through activation of immuno-inflammatory, oxidative and nitrosative stress, and tryptophan catabolite pathways
Version 2 2024-05-30, 15:33
Version 1 2014-10-28, 09:59
journal contribution
posted on 2024-05-30, 15:33 authored by G Anderson, M Maes, Michael BerkMichael BerkSchizophrenia is primed for an increased expression of depression through activation of immuno-inflammatory, oxidative and nitrosative stress, and tryptophan catabolite pathways
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Journal
Progress in Neuro-Psychopharmacology and Biological PsychiatryVolume
42Pagination
101-114Location
EnglandISSN
0278-5846eISSN
1878-4216Language
EnglishPublication classification
C1 Refereed article in a scholarly journalPublisher
PERGAMON-ELSEVIER SCIENCE LTDPublication URL
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Science & TechnologyLife Sciences & BiomedicineClinical NeurologyNeurosciencesPharmacology & PharmacyPsychiatryNeurosciences & NeurologyCytokinesDepressionInflammationOxidative stressSchizophreniaTryptophanMESSENGER-RNA EXPRESSIONSINGLE-NUCLEOTIDE POLYMORPHISMSINDOLEAMINE 2,3-DIOXYGENASE IDORECENT-ONSET SCHIZOPHRENIACELL-MEDIATED-IMMUNITYVITAMIN-D DEFICIENCYMU-OPIOID RECEPTORSN-ACETYL CYSTEINEMAJOR DEPRESSIONNITRIC-OXIDE970111 Expanding Knowledge in the Medical and Health Sciences110319 Psychiatry (incl Psychotherapy)111714 Mental Health920410 Mental HealthInnovation in Mental and Physical Health and Clinical TreatmentSchool of MedicineFaculty of Health
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