miR-23a suppression accelerates functional decline in the rNLS8 mouse model of TDP-43 proteinopathy
Version 2 2024-06-04, 03:43Version 2 2024-06-04, 03:43
Version 1 2021-11-22, 08:07Version 1 2021-11-22, 08:07
journal contribution
posted on 2024-06-04, 03:43 authored by S Tsitkanou, PA Della Gatta, Gavin AbbottGavin Abbott, MA Wallace, A Lindsay, F Gerlinger-Romero, AK Walker, VC Foletta, Aaron RussellAaron RussellmiR-23a suppression accelerates functional decline in the rNLS8 mouse model of TDP-43 proteinopathy
History
Journal
Neurobiology of DiseaseVolume
162Article number
ARTN 105559Pagination
1-15Location
Amsterdam, The NetherlandsOpen access
- Yes
Link to full text
ISSN
0969-9961eISSN
1095-953XLanguage
EnglishPublication classification
C1 Refereed article in a scholarly journalPublisher
ElsevierUsage metrics
Keywords
ALSAmyotrophic lateral sclerosisAMYOTROPHIC-LATERAL-SCLEROSISbeta-AmyloidDEGENERATIONDIFFERENTIATIONEXERCISELife Sciences & BiomedicineMICRORNA-23AmiR-23aMotor neuron diseaseMyogenic regulatory factorsNeurosciencesNeurosciences & NeurologyRESISTANCErNLS8 miceScience & TechnologySkeletal muscleSKELETAL-MUSCLESUPEROXIDE-DISMUTASESURVIVALTDP-433209 Neurosciences
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